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Being an Evening Person Isn’t the Problem

Being an Evening Person Isn’t the Problem

by Jumana Antoun in News

You know the feeling. It’s 10:00 PM, and while the rest of the world is winding down, your brain is just hitting its stride. You’re sharp, creative, and finally "awake." But then 6:00 AM rolls around, and the alarm clock feels like a personal assault.

For many, the struggle with obesity isn't a lack of willpower, it’s a state of internal desynchrony. If you are a "Night Owl" (Evening Chronotype) living in a "Morning Lark" world, you are likely suffering from a biological mismatch that actively sabotages your metabolism. It’s time to start understanding your Chronotype.

 Why do "Night Owls" even exist?

Chronotype is a heritable, polygenic trait, meaning your preference for mornings or nights is partially "hardwired" into your DNA through variations in specific clock genes. In large modern population samples, most people fall in the middle, with smaller minorities at the extremes (“definitely morning” or “definitely evening”). For example, in UK Biobank (a community-based cohort with 439,933 adults aged ~40–69), chronotype assessed by a standard self-rating question showed 27.1% “definitely morning,” 63.9% intermediate (split across “more morning” and “more evening”), and 9.0% “definitely evening”. The distribution shifts with age (later during adolescence/early adulthood, earlier with aging) and differs with environment and social constraints, but the “mostly intermediate, fewer extremes” pattern remains consistent.

Being a Night Owl might have saved your ancestors' lives. The Hadza are a conteporary hunter-gatherer community in northern Tanzania whose traditional lifestyle, free from the distortions of electric light and rigid industrial schedules, provides researchers with a vital "natural window" into how human sleep and circadian rhythms evolved. Researchers studying the Hadza hunter-gatherers found that in a typical camp, someone was awake nearly 99.8% of the time. While the "Larks" (morning people) slept, the "Owls" (night people) stayed awake, acting as sentinels to guard against predators. Your late-night energy isn't a "glitch"—it was once a survival gift. The problem isn't your clock; it's that we now live in a world that runs exclusively at "Lark Time."

Social Jetlag

Productivity culture quietly assigns moral value to waking at dawn. The early riser is praised, and the night person is treated as the one who needs “fixing.”  The “problem” isn’t being a night person at all—it’s being a night person in a world that runs on morning rules. The problem isn't being an Owl; it's Social Jetlag—the discrepancy between your biological clock and your social obligations (work/school). Your Chronotype (evening person) is basically your body’s timing preference—when sleep feels easiest, when alertness rises, when your internal night begins. One-way researchers measure that internal timing is DLMO (dim-light melatonin onset), the point when your brain begins secreting melatonin under dim light. Later chronotypes tend to have later DLMO on average—meaning their biological night starts later.

Your circadian rhythm is your body’s built-in 24-hour timing system. Think of it as a conductor that keeps thousands of “daily programs” coordinated: when you feel sleepy vs alert, when hormones rise and fall, when digestion is primed, when body temperature peaks, even when immune cells are more active. The master timekeeper sits in the brain, in the suprachiasmatic nucleus (SCN) of the hypothalamus. The SCN gets direct input from the eyes (special light-sensitive retinal cells), so light is the strongest cue that resets the clock each day. From there, the SCN synchronizes the rest of the body through nerves, hormones, and behavior—helping keep peripheral clocks in organs like the liver, pancreas, fat, and gut aligned with the day–night cycle. Melatonin rises in the evening to signal “biological night,” while cortisol typically rises toward morning to support wakefulness and energy mobilization.

Health effects

When an "Owl" is forced to wake up at 6:00 AM, the Brain Clock: Says it’s midnight and time for rest. The Social Clock: Says it’s 6:00 AM and time for school or work. They will experience social jetlag. This isn't just about being tired; it’s a total internal desynchrony leading to health consequences.

 Research consistently shows that the evening chronotype is tied to a higher body mass index (BMI) and a more "obesogenic" lifestyle, but the reason isn't necessarily found in the "Owl" genes themselves. Instead, the risk is driven by a series of behavioral shifts that occur when your biological timing is pushed late: shorter sleep, late-night light exposure, and irregular eating.  In studies of Mediterranean populations, "late eaters" lost significantly less weight than "early eaters," even when their total calorie intake and physical activity were identical. Late eaters also tended to be more evening-type and more likely to skip breakfast.  This suggests that eating when your peripheral clocks (in the liver and gut) are winding down makes you metabolically inefficient. Eating a late-night "second dinner" happens when your pancreas is "asleep," leading to poor glucose control and increased fat storage. Misalignment blunts the morning cortisol surge and disrupts appetite hormones (Leptin and Ghrelin), making you crave high calorie, "ultra-processed" foods at night.

When your sleep schedule swings by more than two hours between your work week and your weekend, you aren't just tired; you are statistically 1.3 to 1.7 times more likely to struggle with obesity. For the typical "Owl," this often happens. Furthermore, definite evening" types face a 15–25% higher hazard of cardiovascular events and 1.5 to 1.8 times higher odds of depression, and a BMI that is 0.3 to 0.6 kg/m² higher than their morning-oriented peers.

A lot of studies link eveningness to cardiometabolic and mental-health risks.  Much of the observed risk is plausibly driven by repeated exposure to sleep restriction, circadian phase misalignment, and behavior timing shifts (late eating, reduced morning light, inconsistent activity), rather than the chronotype trait itself. The takeaway is your chronotype is not a pathology. The harm stems from misalignments, which is a modifiable factor.

If it’s misalignment, what helps?

There is no need to change who you are. Modern life floods evenings with light and often deprives us of morning light. The goal is not to turn an owl into a lark. The goal is alignment. 

1-    Use light strategically. Light is the main clock-reset tool. In a famous natural light experiment, adults who spent one week camping without artificial light shifted their circadian timing earlier — especially those who were later chronotypes. Light expands or shrinks the owl-lark gap. Get 15–30 minutes of outdoor sunlight as soon as you wake up which can help "pull" your rhythm earlier, making it easier to fall asleep at night. Moreover, your brain thinks the blue light from your phone is the sun. Dim your lights and use filters after 8:00 PM to allow melatonin to rise naturally.

2-    Stabilize wake time. The biggest mistake is "catching up" on sleep by 4 hours on the weekend. This creates a permanent state of jetlag. Try to keep your weekend wake time within 60 minutes of your weekday alarm.

3-    Watch your eating habits. If you’re an evening type, evenings are often your highest-risk window for cravings. Plan it: a structured high-protein snack before cravings hit, a “kitchen closing” routine or a default if-then plan (“If I want something at 10 pm → I have X.”)

 “Don’t eat late” is not about 8 p.m. or 9 p.m. It’s about your biological night. Your body shifts into night mode when melatonin — your sleep hormone — starts rising. For most people, that happens somewhere between 8:00 and 10:30 p.m., but it depends on your chronotype. When you eat close to that internal night, your body handles glucose less efficiently. Blood sugar rises higher. Insulin works less effectively. The same meal eaten earlier in your biological day produces a better metabolic response. Finish your last main meal 2–3 hours before your natural bedtime. Keep your evening timing consistent throughout the week.

Conclusion

Chronotypes likely exist for deep-seated biological and evolutionary reasons—ranging from your genetic "wiring" to the protective "sentinel" roles humans played in ancestral groups. However, modern life, with its early-morning schedules, artificial bright nights, and late-night eating, forces certain chronotypes to pay a much higher physiological price.

The JA Method shifts the conversation away from the frustrating attempt to "fix" your nature. Instead of asking, “How do I force myself to become a morning person?” the focus becomes: “How do I reduce my internal misalignment and protect my highest-risk hours?” By identifying those windows where your biology and your environment clash, you can stop the metabolic friction. That strategic alignment is where real health wins—and sustainable weight management—actually live.

References

  1. Arab, A., Karimi, E., Garaulet, M., & Scheer, F. A. J. L. (2024). Social jetlag and obesity: A systematic review and meta-analysis. Obesity Reviews, 25(3), e13664. https://doi.org/10.1111/obr.13664
  2. Garaulet, M., Gómez-Abellán, P., Alburquerque-Béjar, J. J., Luján-Antequera, S., Ordovás, J. M., & Scheer, F. A. J. L. (2013). Timing of food intake predicts weight loss effectiveness. International Journal of Obesity, 37(4), 604–611. https://doi.org/10.1038/ijo.2012.229
  3. Jones, S. E., Lane, J. M., Wood, A. R., van Hees, V. T., Tyrrell, J., Beaumont, R. N., Jeffries, A. R., Dashti, H. S., Hillsdon, M., Ruth, K. S., Tuke, M. A., Yaghootkar, H., Sharp, S. A., Jie, Y., Thompson, W. D., Harrison, J. W., Dawes, A. S., Budd, C. L., Lewis, D. A., . . . Weedon, M. N. (2019). Genome-wide association analyses of chronotype in 447,678 individuals identifies new loci and genetic links with self-reported diurnal preference and mental health. Nature Communications, 10(1), 481. https://doi.org/10.1038/s41467-018-08259-7
  4. Kennaway, D. J. (2023). The dim light melatonin onset: What it is and what it is not, and its relationship with morningness/eveningness. Sleep, 46(5), zsad033. https://doi.org/10.1093/sleep/zsad033
  5. Lotti, S., Pagliai, G., Colombini, B., Sofi, F., & Dinu, M. (2022). Chronotype differences in energy intake, dietary quality, and meal timing: A systematic review with meta-analysis of observational studies. Advances in Nutrition, 13(6), 2239–2252. https://doi.org/10.1093/advances/nmac075
  6. Malone, S. K., Patterson, F., Lu, Y., Lozano, A., & Hanlon, A. (2016). Ethnic differences in sleep duration and morning–evening type in a population sample. Chronobiology International, 33(1), 10–21. https://doi.org/10.3109/07420528.2015.1107722
  7. Neves, A. R., Albuquerque, T., & Quintela, T. (2022). Circadian rhythm and disease: Relationship, new insights, and future perspectives. Journal of Cellular Physiology, 237(10), 3737–3758. https://doi.org/10.1002/jcp.30815
  8. Parsons, M. J., Moffitt, T. E., Gregory, A. M., Goldman-Mellor, S., Nolan, P. M., Poulton, R., & Caspi, A. (2015). Social jetlag, obesity and metabolic disorder: Investigation in a cohort study. International Journal of Obesity, 39(5), 842–848. https://doi.org/10.1038/ijo.2014.201
  9. Kianersi, S., Potts, K. S., Wang, H., Sofer, T., Noordam, R., Rutter, M. K., ... & Huang, T. (2026). Chronotype, Life’s Essential 8, and risk of cardiovascular disease: a prospective cohort study in UK Biobank. Journal of the American Heart Association, 15(3), e044189.
  10. Wright, K. P., McHill, A. W., Birks, B. R., Griffin, B. R., Rusterholz, T., & Chinoy, E. D. (2013). Entrainment of the human circadian clock to the natural light-dark cycle. Current Biology, 23(16), 1554–1558. https://doi.org/10.1016/j.cub.2013.06.039
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Food flexibility: Secret to Lasting Weight Loss

Food flexibility: Secret to Lasting Weight Loss

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If parents accommodated selective eating or avoided challenging food preferences, those patterns often persisted into adulthood.- Sensory sensitivity: Some people have heightened responses to textures, tastes, or smells. This can make certain foods feel overwhelming or unpleasant.- Associations and memories: Negative experiences with food (like being forced to eat something disliked or linking a food to illness) can create lasting aversions.- Psychological links: Studies suggest picky eating can be associated with anxiety, obsessive traits, rigidity, or a need for control, which makes food exploration more challenging. The good news is that most picky eating is flexible — with curiosity, repeated exposure, and small experiments, adults can expand their food variety over time. Over time, the unfamiliar becomes familiar, and the “disliked” can shift into “tolerable” or even “enjoyable.” When Picky Eating Becomes a Disorder It’s important to distinguish everyday picky eating from a clinical condition known as Avoidant/Restrictive Food Intake Disorder (ARFID). ARFID goes beyond selectiveness: it involves extreme restriction of foods due to sensory sensitivities, fear of adverse consequences (like choking or vomiting), or a lack of interest in eating. Unlike anorexia or bulimia, ARFID is not driven by body image concerns but by anxiety and avoidance around food itself.Adults with ARFID may experience significant health consequences, including nutrient deficiencies, weight loss, and social withdrawal. Treatment often requires professional support, such as cognitive-behavioral therapy and structured exposure to new foods. Most picky eating, however, is not ARFID. This distinction is important: while ARFID needs intensive therapy, everyday picky eating can be addressed through gradual, self-guided change. Why You Need Food Flexibility in Weight Loss When you’re trying to lose weight, it’s not enough to simply cut portions or reduce snacking. The challenge is that picky eating often limits access to these nutrient-dense foods. If you avoid beans, lentils, fish, eggs, or vegetables because of taste or texture, you may unintentionally cut yourself off from the very foods that make a calorie deficit sustainable. That’s why building food flexibility is not just about variety for its own sake—it's about ensuring your diet includes the protein and fiber that reduce hunger, stabilize energy, and make long-term weight management possible. Research consistently shows that protein increases satiety and helps preserve lean muscle mass during weight loss, which is critical for maintaining metabolic health. 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The first bite may feel uncomfortable, but with repetition, the unfamiliar becomes familiar. The lesson is simple: not every food has to be a favorite or delicious. Some will be “just fine,” and that’s perfectly acceptable. If a food is truly aversive, don’t force it. But if it’s tolerable, even if not delightful, consider including it for its nutritional value. Each time you stretch your comfort zone, you prove to yourself that you can adapt—and that adaptability is the foundation of lasting change. Final Thoughts Weight loss isn’t just about shrinking portions or dropping snacks—it’s about freedom and expanding possibilities. The goal is not to force yourself to eat something you truly dislike. The goal is to become curious. By cultivating food flexibility, you’re not only nourishing your body but also strengthening your mindset. Each new flavor, texture, and experience is proof that you can adapt, grow, and thrive. References Dial, L.A., Jordan, A., Emley, E., Angoff, H.D., Varga, A.V. and Musher-Eizenman, D.R., 2021. Consequences of picky eating in college students. Journal of Nutrition Education and Behavior, 53(10), pp.822-831.https://doi.org/10.1016/j.jneb.2021.07.00. https://doi.org/10.1093/ajcn/nqy308 Kauer J, Pelchat ML, Rozin P, Zickgraf HF. Adult picky eating. Phenomenology, taste sensitivity, and psychological correlates. Appetite. 2015 Jul;90:219-28. doi: 10.1016/j.appet.2015.03.001. Epub 2015 Mar 5. PMID: 25747855; PMCID: PMC8477986. Matthews S. Health Coaching as an Intervention for Picky Eaters. American Journal of Lifestyle Medicine. 2020 Jul 29;14(6):606-611. doi: 10.1177/1559827620943821. PMID: 33117103; PMCID: PMC7566187. Spill, M.K., Johns, K., Callahan, E.H., Shapiro, M.J., Wong, Y.P., Benjamin-Neelon, S.E., Birch, L., Black, M.M., Cook, J.T., Faith, M.S. and Mennella, J.A., 2019. Repeated exposure to food and food acceptability in infants and toddlers: a systematic review. The American Journal of Clinical Nutrition, 109, pp.978S-989S. Zickgraf, H.F., Franklin, M.E. & Rozin, P. Adult picky eaters with symptoms of avoidant/restrictive food intake disorder: comparable distress and comorbidity but different eating behaviors compared to those with disordered eating symptoms. Journal of Eating Disorders 4, 26 (2016). https://doi.org/10.1186/s40337-016-0110-6 Zickgraf, H.F. and Schepps, K., 2016. Fruit and vegetable intake and dietary variety in adult picky eaters. Food Quality and Preference, 54, pp.39-50. https://doi.org/10.1016/j.foodqual.2016.06.012  

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Why Having ‘Healthy Obesity’ Is Still Dangerous for Your Health?

Why Having ‘Healthy Obesity’ Is Still Dangerous for Your Health?

Introduction Obesity has long been linked to shorter lifespans, lower quality of life, and increased risk of disabilities due to cardiovascular diseases like diabetes and atherosclerosis. This raised the question: how can some individuals with obesity appear healthy over the long term, whereas others show immediate signs of health problems? The concept of metabolically healthy obesity has caused quite a stir in the medical community and beyond in recent years, as it challenges deeply held beliefs and long-standing understandings. But the recognition of this phenomenon is far from a new discovery! It dates to Jean Vague’s observations in the 1950s that certain individuals with obesity had different predispositions towards diabetes and atherosclerosis, which seemed to be related to where their fat was concentrated on their bodies. What is the definition of metabolically healthy obesity? While individuals who have metabolically healthy obesity (MHO) have been extensively researched, a consensus on a definition in adults has only recently been reached. Metabolically healthy obesity (MHO) is defined as a BMI of 30 kg/m2 or higher and no concurrent metabolic disorder or cardiovascular diseases, such as type 2 diabetes or atherosclerotic disease. A more recent definition has been proposed, requiring additional criteria such as serum triglycerides ≤150 mg/dl and HDL-cholesterol serum concentrations higher than 40 mg/dl in men or 50 mg/dl in women. Do people with “healthy obesity” exist? Recent studies show that MHO is found in 35% of the world’s population. However, there were notable differences between regions and various parts of the world. Irrespective of the definitions used and the remarkable regional and gender variation, MHO is not a rare condition. Long-term studies have suggested that MHO might be a transient state. About 30% of people with MHO develop or convert to a metabolically unhealthy obesity (MUHO) state with cardio-metabolic complications over a 5-10-year period. Aging and additional weight gain are risk factors for this conversion. Fortunately, increasing physical activity and losing weight can increase the likelihood of preserving MHO status. What are the health risks of “healthy obesity”? Metabolically healthy obesity is an interesting and complex phenomenon – on the one hand, these individuals are typically free of metabolic signs associated with “unhealthy weight”, yet they still have higher cardiovascular risks than those who maintain a normal weight.  However, their cardiovascular risks are lower than those of people with metabolically unhealthy obesity (MUHO). A 2019 study, for example, found that those with metabolically unhealthy obesity (MUHO) are 5-20 times more likely to develop diabetes than those with a ‘metabolically healthy normal weight’. Even for those with metabolically healthy obesity (MHO), there is still a 4-fold increased risk of developing diabetes compared to metabolically healthy normal weight. Similarly, the risk of cardiovascular diseases is 50% greater in people with MHO at baseline than in people who are metabolically healthy at a normal weight.  This means that while there are still risks involved with being classified as metabolically healthy obese, they are nowhere near as severe as those associated with those who are metabolically unhealthy. How to prevent the transition to metabolically unhealthy obesity? As MHO is not a static condition, attention has now shifted to what factors may predict metabolic deterioration from MHO to MUHO. A study in Spain identified three main factors: an increase in BMI, waist circumference, and waist-to-hip ratio. On the flip side, lifestyle choices like following a healthy diet, regular exercise, and avoiding or quitting smoking prevented the transition to MUO. It’s clear from this that good habits can help maintain our health and protect us from potentially serious conditions like MUO. Conclusion There is such a thing as metabolically healthy obesity, but it may be transient and comes with its own risks. The best way to avoid these risks is to maintain a healthy weight through diet and exercise. So, let’s take care of our health and be more mindful of what we eat and how much we exercise. References Blüher, M. (2020). Metabolically Healthy Obesity. Endocrine Reviews, 41(3), 405.‏ Smith GI, Mittendorfer B, and Klein S. 2019. Metabolically healthy obesity: facts and fantasies. The Journal of clinical investigation 129:3978-3989. Post navigation

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Homeostatic and Hedonic Hunger

Homeostatic and Hedonic Hunger

Have you ever wondered why you can’t stop eating, even when you’re full? Or why do you eat more than your body needs? Have you ever felt like starving even after eating a big meal? Or found yourself craving food, even if it wasn’t remotely close to mealtime? If so, what you experienced is called hedonic hunger. It turns out that what we think of as “hunger” is two different things. In modern times, among well-nourished populations, an increasing proportion of human food consumption appears to be driven by pleasure, not the need for calories —which isn’t necessarily bad! In this article, we’ll look at homeostatic and hedonic hunger to better understand the motivations behind food consumption. History of Human Food Consumption For most of human history and prehistory, the primary objective of seeking food was survival through maintaining energy homeostasis and avoiding starvation. Humans had to eat enough food that provided enough calories to meet their daily needs. It was essential for early humans to have enough energy to perform physical tasks such as gathering food and other resources or defending themselves from predators. In modern times, energy homeostasis is no longer an issue for most people living in developed countries who no longer need to worry about finding food for existence. The availability of an abundance of tasty foods has changed our relationship with eating from one primarily focused on survival to one also driven by pleasure. Hemeostatic vs. Hedonic Hunger Homeostatic hunger is what most people think of when they hear the word “hunger.” It refers to the body’s need for energy to maintain balance (homeostasis). When your body needs energy, it sends signals to your brain telling you to eat. Physiological needs drive this type of hunger. Hedonic hunger, however, is driven by pleasure and anticipation of reward rather than physiological needs. Hedonic hunger is an intense, psychological desire for food triggered by external cues such as the sight or smell of food, social events, time, or advertising campaigns. Unlike homeostatic hunger, which is generally limited to only what our bodies need in terms of calories, hedonic hunger is not constrained by physical limitations and can often result in over-consumption. The interplay between these two types of hunger has become increasingly complex in recent years due to the availability of calorie-dense foods with high levels of added sugar and fat. These foods are designed for maximum pleasure, making them difficult for many people to resist even if their body does not need them.   How do We Know We are Hungry? Hunger is challenging to specify. We need help not only quantifying our hunger, but many need help identifying whether we are hungry at all. There may be no solid characteristic pattern of hunger symptoms when it comes to typical hunger sensations. When participants were asked about hunger sensations, there were three key identifiers. First and foremost, most people experience a growling stomach as an indicator that it’s time for food. Secondly, many people also report feeling an empty sensation in their stomach when they are hungry. Lastly, some people describe feeling mild to moderate stomach pain when hungry. In addition to these typical hunger sensations, many people experience extreme hunger sensations: headaches, lightheadedness, or dizziness, being fixated on food and feeling irritable or cranky. Understanding the distinction between homeostatic and hedonic hunger is essential for physical and mental health since it can help us make better food choices. There are distinct neural pathways associated with homeostatic and hedonic motivation for eating with animal models;  however, further research should be conducted to understand how these systems interact within humans. It is sometimes hard to recognize the type of hunger in real life. However, hedonic hunger involves two constructs: liking and wanting. Liking vs. Wanting Food The primary difference between liking and wanting food lies in the motivation behind eating. Liking is a pleasure derived from oro-sensory stimulation of food – think flavors, textures, smells, etc. Conversely, wanting is defined as incentive salience or the motivation to engage in eating. When you want something, you are more likely to seek it out despite any associated costs or calories involved. By understanding these two different types of motivation when it comes to eating, we can begin to make better decisions about our diets and health goals. For example, if you “like” a specific type of food but do not necessarily need it (i.e., don’t have an urgent “want”), then it may be best to practice moderation rather than overindulge in something your body doesn’t need. On the other hand, if you want a particular type of snack despite having already eaten earlier in the day (or week!), then perhaps it’s time to take a step back and ask yourself why – are you bored? Stressed? Anxious? Identifying those underlying motivations for wanting specific foods can help us develop healthier habits and make more informed decisions about what we eat. Conclusion Enhanced hedonic response to foods can augment hunger even when satiated, meaning that even if we don’t feel hungry, we may still be tempted by tasty treats. Identifying between homeostatic and hedonic hunger can be challenging but ultimately rewarding. You will have more control over your eating habits if you understand where those cravings come from! By paying attention to cues from your body, keeping track of meals/snacks throughout the day, and taking breaks whenever necessary, you’ll be able to tell which type of hunger is driving your appetite–and make healthier choices accordingly! All this shows just how complex our relationship with food can be. References Lowe, M.R. and Butryn, M.L., 2007. Hedonic hunger: a new dimension of appetite?. Physiology & behavior, 91(4), pp.432-439. Blundell, J.E. and Finlayson, G., 2004. Is susceptibility to weight gain characterized by homeostatic or hedonic risk factors for overconsumption?. Physiology & behavior, 82(1), pp.21-25.

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